By J. T. Van Oirschot (auth.), B. Liess (eds.)
The heritage of study on hog cholera (HC)/classical swine fever (CSF) might be approximately divided into 3 levels that are characterised through the tools to be had on the time for demonstrati ng the causati ve agent. section lined the interval earlier than the viral etiology of HC was once chanced on through de Schweinitz and Dorset (1904)*. Thereafter (Phase II) the detection of HC virus (HCV) used to be complete by means of arduous, time-consuming and dear pig inoculation experiments. This explains the vast look for tools not just for detection but additionally for exact infectivity titration in addition to for appropriate serological strategies to unravel pressing difficulties in regards to the pathogenesis, analysis, epidemiology and prophylaxis of HC. It was once now not ahead of the overdue fifties that HC study entered part III while fluorescent antibody innovations provided not just the ability for detection and titration of HCV in porcine mobile cultures but in addition for extra extensive study on hog cholera and its virus. And but, there are various inquiries to be responded, e. g. at the genetic and antigenic relation of HCV to bovine viral diarrhea (BVD) virus. There are symptoms that section IV of HC study will undergo the stamp of biotechnology. In view of this improvement it sounds as if applicable to provide an up-dating and summarizing account of HC/CSF together with comparative facets of infections brought on by structurally comparable viruses. The version of the current quantity may were most unlikely with no the cooperation of a number of recognized scientists who immediately agreed while requested for contribution.
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Extra resources for Classical Swine Fever and Related Viral Infections
Zbl Vet Med B (11 ) : 119-134,1964. 10. Fuchs F: Schweinepest. Handbuch der Virusinfektionen bei Tieren. Band III/I (Ed. RBhrer H), Gustav Fischer Verlag Jena, pp 1-250,1968. 11. Korn G: Uber die VerHnderung von Krankheitsverlaufsformen der Schweinepest und damit verbundene VirulenzverHnderungen des Virus nach Stimulierung der "Lymphozyten". Ber1 MUnch Tierfutzl Wschr ( 86 ) : 447449,1973. 12. Dunne HW: Hog Cholera. In: Diseases of swine (Eds. Dunne HW, Leman AD), Iowa state Univ Press, pp 189-255,1975 13.
II. Irmnunopathological finding in the fetus. Zbl Vet Med B (27) :243252,1980. 75. Done JT, Harding JW: An experimentally-induced congenital tremor of pigs. Exc Med Int Congr Ser (173):112-118,1969. 76. Vannier P, Plateau E, Tillon JP: Congenital tremor in pigs farrowed fran sows given hog cholera virus during pregnancy. Am J Vet Res (42):135-137,1981. 77. Meyer H, Liess B, Frey HR, Hermanns W, Trautwein G: Experimental transplacental transmission of hog cholera virus in pigs. IV. Virological and serological studies in newborn piglets.
Hog cholera in gnotobiotic pigs. Clinical signs and gross pathologic findings in germ-free and monocontaminated pigs. J. Amer. Vet. Med. Ass. 140: 19561961,1962. 7. Matthias, D. und Klaus, H. Pathologisch-physiologische Untersuchungen zum Hamoglobinund Eisenstoffwechsel beiSchweinepest. Arch. expo Vet. Med. 12: 427-443, 1958. 8. J. W. Focal necrosis in the mucosa of the gall bladder in pigs with hog cholera. Amer. J. Vet. Res. 22: 391-395, 1961. 9. M. A. Bone changes in pigs infected with hog cholera.
Classical Swine Fever and Related Viral Infections by J. T. Van Oirschot (auth.), B. Liess (eds.)
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